著者

廣瀬 聰明 野中 伸介 上野 栄和 木村 重治 吉本 正太 道家 孝幸 杉 憲 岡村 健司

出版者

日本肩関節学会

雑誌

肩関節 (ISSN:09104461)

巻号頁・発行日

vol.35, no.3, pp.883-887, 2011 (Released:2011-12-21)

参考文献数

12

被引用文献数

4

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We performed arthroscopic rotator cuff repair (ARCR) for all rotator cuff tears. The purpose of this study was to evaluate the clinical results of ARCR using double-row technique. We retrospectively studied 64 patients (65 shoulders) who had received ARCR using double-row technique and who were followed up for more than 2 years. The patients were 30 males and 35 females. The mean age at operation was 65 years old (range, 44-86). The mean postoperative follow-up period was 25 months (range, 24-36). The clinical results were assessed using JOA scores and MRI by Sugaya's classification. Tear size was small tear in 9 shoulders, medium in 36, large in 12, and massive in 8. The mean JOA total score was significantly improved from 66 points preoperatively to 96 points postoperatively. Postoperative MRIs showed 20% re-torn cuff in all cases, especially, 40% in large and massive tears. In 45 shoulders which had MRI taken regularly, re-tear by MRI was revealed within 3weeks: none, at 3 months: 4 shoulders, at 6 months: 1shoulder, at 1 year: 4 shoulders, and 2 years: none. In this study, the clinical results of ARCR using double-row technique was mostly satisfactory. But JOA score in no tear group (97points) was better than re-tear group (92points). So we have to consider the methods to prevent re-tear after ARCR.

著者

射場 浩介 道家 孝幸 花香 恵 金谷 久美子 阿部 恭久 山下 敏彦

出版者

日本疼痛学会

雑誌

PAIN RESEARCH (ISSN:09158588)

巻号頁・発行日

vol.31, no.4, pp.203-209, 2016-12-26 (Released:2017-01-27)

参考文献数

20

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We have recently demonstrated that pathological changes leading to increased bone resorption by osteoclast activation are related to the induction of pain–like behavior in ovariectomized (OVX)mice. In addition,we have shown that the skeletal pain accompanying osteoporosis is possibly associated with the acidic microenvironment caused by osteoclast activation under a high bone turnover state. We, therefore, hypothesize that another osteoporosis model mouse might reveal the induction of pain–like behaviors in relation with osteoporotic changes. In this study, we demonstrated that regional osteoporosis of hind limbs induced pain–like behaviors using tailsuspended mice as another osteoporosis model.The hind limbs of tail–suspended mice were unloaded for 2 weeks, during which time the mice revealed significant regional osteoporotic changes in their hind limbs accompanied by osteoclast activation. In addition, these changes were significantly recovered by the resumption of weight bearing on the hind limbs for 4 weeks. Consistent with the pathological changes in the hind limbs, pain–like behaviors in the mice were induced by tail suspension and recovered by the resumption of weight bearing. Moreover, treatment with bisphosphonate significantly prevented the triggering of the regional osteoporosis and pain–like behaviors, and antagonists of the acid–sensing nociceptors, such as transient receptor potential channel vanilloid subfamily member 1 and acid–sensing ion channels, significantly improved the painlike behaviors in the tail–suspended mice.We, therefore, believe that pathologic changes of osteoporosis due to osteo clast activation might be a trigger for the pain–like behaviors in osteoporosis patients.