4 0 0 0 OA 急冷と準安定平衡
- 著者
- 新宮 秀夫 鈴木 亮輔 石原 慶一
- 出版者
- 公益社団法人 日本材料学会
- 雑誌
- 材料 (ISSN:05145163)
- 巻号頁・発行日
- vol.33, no.366, pp.239-245, 1984-03-15 (Released:2009-06-03)
- 参考文献数
- 22
- 被引用文献数
- 1 2
1 0 0 0 OA エチオピア西南部における伝統製鉄技術の冶金学的検証
- 著者
- 山末 英嗣 村橋 勲 奥村 英之 石原 慶一
- 出版者
- 公益社団法人 日本金属学会
- 雑誌
- 日本金属学会誌 (ISSN:00214876)
- 巻号頁・発行日
- vol.71, no.9, pp.763-771, 2007 (Released:2007-09-01)
- 参考文献数
- 12
An abandoned traditional steelmaking in Southwest Ethiopia (Dime) has been restored, and the technology and knowledge used for the operation have been metallurgically analyzed. The restoration was carried out with local blacksmiths from September to October in 2004, and the steelmaking operation including mining, construction of a furnace and charcoal production, etc was successfully performed. Produced sponge iron contains 0.31∼0.48 mass% carbon without any impurities. The yield ratio of the iron was about 40%. The collected slag contains the elements of Fe, Si, Al, K, P, Ti and Mn, which are typical components of slag. The blacksmiths used three kinds of iron ore, named “Balt”, “Bullo” and “Gachi”. The former two ores mainly consist of goethite (α-FeO(OH)) and kaolinite (Al2O3•2SiO2•2H2O). The latter includes calcium phosphate hydrate (Ca3(PO4)2•xH2O) in its white part, as well as the goethite and kaolinite. The reason why the local blacksmiths specifically selected “Gachi” as the best ore for their steelmaking was discussed from the viewpoint of slag forming ability.
1 0 0 0 OA 日本、タイ、ベトナムにおける農村への再生可能エネルギー導入
- 著者
- 石原 慶一
- 出版者
- 研究・イノベーション学会
- 雑誌
- 年次学術大会講演要旨集
- 巻号頁・発行日
- vol.32, pp.134-135, 2017-10-28
一般講演要旨
1 0 0 0 OA 創薬標的の同定を目的としたダウン症マウス脳での変動分子の網羅的解析
- 著者
- 石原 慶一
- 出版者
- 公益社団法人 日本薬学会
- 雑誌
- YAKUGAKU ZASSHI (ISSN:00316903)
- 巻号頁・発行日
- vol.137, no.7, pp.807-810, 2017 (Released:2017-07-01)
- 参考文献数
- 8
Down syndrome, caused by the triplication of human chromosome 21, is the most frequent genetic cause of mental retardation. Mice with a segmental trisomy for mouse chromosome 16, which is orthologous to human chromosome 21, exhibit abnormalities similar to those in individuals with Down syndrome and therefore offer the opportunity for a genotype-phenotype correlation. In the current review, I present several mouse lines with trisomic regions of various lengths and discuss their usefulness for elucidating the mechanisms underlying Down syndrome-associated developmental cognitive disabilities. In addition, our recent comprehensive study attempting to identify molecules with disturbed expression in the brain of a mouse model of Down syndrome in order to develop a pharmacologic therapy for Down syndrome is described.