文献一覧: 00278424 (ISSN) - Ceek.jp Altmetrics

5 0 0 0 OA Form of an evolutionary tradeoff affects eco-evolutionary dynamics in a predator-prey system

著者: Kasada M. Yamamichi M. Yoshida T.
出版者: National Academy of Sciences
雑誌: Proceedings of the National Academy of Sciences (ISSN:00278424)
巻号頁・発行日: vol.111, no.45, pp.16035-16040, 2014-11-11
被引用文献数: 57

遺伝的多様性の新しい影響を発見 -わずかな性質の違いが生態系を変化させる可能性-. 京都大学プレスリリース. 2014-10-23.

2022-10-06 06:47:53
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5 0 0 0 OA Field-induced superconducting phase of FeSe in the BCS-BEC cross-over

著者: Kasahara S. Watashige T. Hanaguri T. Kohsaka Y. Yamashita T. Shimoyama Y. Mizukami Y. Endo R. Ikeda H. Aoyama K. Terashima T. Uji S. Wolf T. von Lohneysen H. Shibauchi T. Matsuda Y.
出版者: National Academy of Sciences
雑誌: Proceedings of the National Academy of Sciences (ISSN:00278424)
巻号頁・発行日: vol.111, no.46, pp.16309-16313, 2014-11
被引用文献数: 315

「ボース・アインシュタイン凝縮に最も近い超伝導状態」を発見. 京都大学プレスリリース. 2014-11-05.

2018-06-22 21:56:00
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4 0 0 0 OA Differentiation-defective phenotypes revealed by large-scale analyses of human pluripotent stem cells.

著者: Koyanagi-Aoi Michiyo Ohnuki Mari Takahashi Kazutoshi Okita Keisuke Noma Hisashi Sawamura Yuka Teramoto Ito Narita Megumi Sato Yoshiko Ichisaka Tomoko Amano Naoki Watanabe Akira Morizane Asuka Yamada Yasuhiro Sato Tosiya Takahashi Jun Yamanaka Shinya
出版者: National Academy of Sciences
雑誌: Proceedings of the National Academy of Sciences of the United States of America (ISSN:00278424)
巻号頁・発行日: 2013-11-20
被引用文献数: 190

大規模解析により品質の悪い多能性幹細胞の見分け方を開発. 京都大学プレスリリース. 2013-11-19.

2014-01-02 07:26:04
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3 0 0 0 OA TCF1 and LEF1 act as T-cell intrinsic HTLV-1 antagonists by targeting Tax.

著者: Ma Guangyong Yasunaga Jun-Ichirou Akari Hirofumi Matsuoka Masao
出版者: National Academy of Sciences
雑誌: Proceedings of the National Academy of Sciences of the United States of America (ISSN:00278424)
巻号頁・発行日: vol.112, no.7, pp.2216-2221, 2015-02-02
被引用文献数: 24

ヒトT細胞白血病ウイルス1型の感染特異性の解明に成功 -HTLV-1感染症の病態解明に期待-. 京都大学プレスリリース. 2015-02-05.

2015-02-06 09:15:44
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2 0 0 0 OA Anomalous superfluid density in quantum critical superconductors

著者: Hashimoto Kenichiro Mizukami Yuta Katsumata Ryo Shishido Hiroaki Yamashita Minoru Ikeda Hiroaki Matsuda Yuji Schlueter John A. Fletcher Jonathan D. Carrington Antony Gnida Daniel Kaczorowski Dariusz Shibauchi Takasada
出版者: National Academy of Sciences
雑誌: Proceedings of the National Academy of Sciences of the United States of America (ISSN:00278424)
巻号頁・発行日: 2013-02-12
被引用文献数: 26

絶対零度の量子ゆらぎにより重くなる超伝導電子 -電子が特定の方向で重くなる異常な現象を発見-. 京都大学プレスリリース. 2013-02-12.

2013-02-20 01:57:15
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2 0 0 0 OA Coordinated regulation of transcription factors through Notch2 is an important mediator of mast cell fate

著者: Sakata-Yanagimoto Mamiko Nakagami-Yamaguchi Etsuko Saito Toshiki Kumano Keiki Yasutomo Koji Ogawa Seishi Kurokawa Mineo Chiba Shigeru
出版者: The National Academy of Sciences of the USA
雑誌: Proceedings of the National Academy of Sciences of the United States of America (ISSN:00278424)
巻号頁・発行日: vol.105, no.22, pp.7839-7844, 2008-06
被引用文献数: 52 33

Mast cells are thought to participate in a wide variety of pathophysiological conditions. Mechanisms of regulation, however, of mast cell production and maturation are still to be elucidated. Mast cell developmental process is likely to be profoundly affected by cell-autonomous transcriptional regulators such as the GATA family and CCAAT/enhancer binding protein (C/EBP) family members. Extracellular regulators such as stem cell factor and IL-3 have essential roles in basal and inducible mast cell generation, respectively. The relationship, however, between the extracellular signaling and cellular transcriptional control is unclear, and the trigger of the mast cell development remains elusive. Notch signaling plays a fundamental role in the lymphopoietic compartment, but its role in myeloid differentiation is less clear. Here, we demonstrate that Notch signaling connects environmental cues and transcriptional control for mast cell fate decision. Delta1, an established Notch ligand, instructs bone marrow common myeloid progenitors and granulocyte-macrophage progenitors toward mast cell lineage at the expense of other granulocyte-macrophage lineages, depending on the function of the Notch2 gene. Notch2 signaling results in the up-regulation of Hes-1 and GATA3, whereas simultaneous overexpression of these transcription factors remarkably biases the progenitor fate toward the mast cell-containing colony-forming cells. C/EBPα mRNA was down-regulated in myeloid progenitors as a consequence of Hes-1 overexpression, in agreement with the recent proposal that the down-regulation of C/EBPα is necessary for mast cell fate determination. Taken together, signaling through Notch2 determines the fate of myeloid progenitors toward mast cell-producing progenitors, via coordinately up-regulating Hes-1 and GATA3.