文献一覧: 久松健一 (著者)

5 0 0 0 IR 原稿の下に隠されしもの--"引用・模倣・盗用・盗作"を通じて文芸の創造のなんたるかを考える(2)「盗むこと」を創作の原理とした男・寺山修司:模倣巡礼年表付

著者: 久松健一
出版者: 明治大学教養論集刊行会
雑誌: 明治大学教養論集 (ISSN:03896005)
巻号頁・発行日: no.446, pp.67-93, 2009-03

2021-03-29 11:45:05
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https://ci.nii.ac.jp/naid/120001941194

3 0 0 0 IR 嚙みつく学者杉本つとむ

著者: 久松健一
出版者: 明治大学図書館
雑誌: 図書の譜 : 明治大学図書館紀要 (ISSN:1342808X)
巻号頁・発行日: no.18, pp.115-125, 2014-03

https://ci.nii.ac.jp/naid/120005479980

2 0 0 0 IR 遠藤周作の秘密(下)

著者: 久松健一
出版者: 明治大学教養論集刊行会
雑誌: 明治大学教養論集 (ISSN:03896005)
巻号頁・発行日: no.408, pp.73-103, 2006-03

2016-04-17 10:24:21
2 + 1 Wikipedia

https://ci.nii.ac.jp/naid/120001441357

1 0 0 0 ロイコトリエンC4,D4及びプロスタグランジンE2の耳管粘液線毛輸送能に与える影響

著者: 岸保鉄也霜村真一久松健一中澤勉中島司野沢出村上嘉彦
出版者: 日本耳科学会
雑誌: Otology Japan (ISSN:09172025)
巻号頁・発行日: vol.4, no.1, pp.90-94, 1994

In middle ear diseases including otitis media with effusion (OME), there are various lipids mediators such as leukotoriene C4, D4 (LTC4, LTD4) and prostaglandin E2 (PGE2) in the effusion of the tympanic cavity. Therefore, it is important to examine the effect of these mediators on middle ear clearance. We studied on the effect of LTC4, LTD4 and PGE2 on mucociliary transport of the eustachian tube both in vitro and in vivo. Healthy guinea pigs with normal Preyer's reflexes were used in vitro study. Normal ciliated epithelium was carefully obtained from the tubotympanium and incubated with RPMI solution in the from of tissue culture. The epithelial specimens were incubated with LTC4, LTD4 and PGE2 respectively, ranged at the concentrations of 10-8M and 10-6M. Ciliated cells of the specimens were observed under an inverted microscope. Ciliary activity of each ciliated cell was photo-electrically measured on a TV monitor. LTC4 and LTD4 inhibited ciliary activity at the concentrations of 10-8M and 10-6M in vitro. PGE2 promoted ciliary activity at the concentrations of 10-8M and 10-6M. Healthy chinchillas were used in vivo study. The animals were free of middle ear infection and hearing loss as determined by otomicroscopy, tympanometry and auditory brainstem response (ABR). One ml each of 10-5M LTC4, LTD4, PGE2 and the control solution (10-3 ethanol/saline) was directly injected into the tympanic bulla with a 27-gauge syringe under anesthetization. The middle ears were examined by otomicroscopy, tympanometry and ABR across time. LTC4 and LTD4 inhibited mucociliary transport of the eustachian tube. However, there was no significant difference between PGE2 and the control.