著者
池崎 和海 芝口 翼 杉浦 崇夫 宮田 浩文
出版者
一般社団法人日本体力医学会
雑誌
体力科学 (ISSN:0039906X)
巻号頁・発行日
vol.66, no.5, pp.345-354, 2017-10-01 (Released:2017-09-29)
参考文献数
27
被引用文献数
1

Although icing treatment has been well accepted as aftercare in sports fields, the detailed mechanisms of the treatment is not fully understood. In this study, we investigated the effect of icing treatment on the recovery process of rat plantaris muscles with artificially induced muscle damage. Sixty male Wistar rats (8-weeks-old) were randomly assigned to three groups; control (CTL), bupivacaine-injected (BPVC), and icing treatment after BPVC (ICE). Icing treatment was applied for 20 min immediately after BPVC, and the treatment was used once per day for 3 days. The plantaris muscles were removed at 3, 7, 15, and 28 days after the muscle damage, then immunohistochemical and real time RT-PCR analysis were performed. In histochemical analysis, although significant changes were found in the relative muscle weight, cross-sectional area of muscle fiber, percentage of muscle fiber with central nuclei, and expressed immature myosin heavy chain isoforms after muscle damage, as compared to the CTL group, no differences were found between BPVC and ICE groups. In mRNA expression analysis, the ICE group had a significantly lower value of MyoD than the BPVC group at 3 days after the damage. Expression of IL-6 mRNA, which relates to muscle inflammation, indicated significantly higher value in BPVC, but not in ICE, than CTL groups at 7days after the damage. Furthermore, BKB2 receptor, which relates to acute muscle soreness, indicated a significantly higher expression in BPVC than ICE groups at 3 days after the damage. These results suggest that icing treatment is effective to suppress muscle inflammation and soreness at an early stage of recovery from damage, but not effective for muscle regeneration at a later stage.
著者
松永 智 佐渡山 亜兵 宮田 浩文 勝田 茂
出版者
一般社団法人日本体力医学会
雑誌
体力科学 (ISSN:0039906X)
巻号頁・発行日
vol.39, no.2, pp.99-105, 1990-04-01 (Released:2010-09-30)
参考文献数
21
被引用文献数
1 2

We investigated the effects of strength training a muscle fiber conduction velocity in biceps brachii of 7 male students. The subjects were trained to exhaustion by 60% of maximum isotonic voluntary contraction with 3 sets/day, 3 days/week for 16 weeks. The muscle fiber conduction velocity was measured with a surface electorode array placed along the muscle fibers, and calculated from the time delay between 2 myoelectric signals recorded during a maximal voluntary contraction. Upper arm girth significantly increased (p<0.01), from 29.2±1.4 cm (means±S. D.) to 30.6±1.5 cm. On the other hand, training induced no significant changes in upper arm skinfold. A significant difference between pre- and post-training was found in maximum isotonic strength (p<0.01) . Although maximum isometric strength showed no significant changes with training, there was a tendency for an increase in maximum isometric strength. Muscle fiber conduction velocity increased by 3.5% during training period, but this was not significant. These results suggest no effects of strength training on muscle fiber conduction velocity.
著者
長久 広 宮田 浩文
出版者
一般社団法人日本体力医学会
雑誌
体力科学 (ISSN:0039906X)
巻号頁・発行日
vol.68, no.6, pp.357-367, 2019-12-01 (Released:2019-12-03)
参考文献数
62

Hypoxic condition of skeletal muscle is caused not only by hypoxia exposure but also by exercise and disease etc. It is thought that clarifying a mechanism of response to hypoxia in vivo is useful for developing better training methods and treatment strategies. However, at present, research dealing with the effects of hypoxic exposure on skeletal muscle have not shown consistent results. Hypoxic exposure results in angiogenesis or muscle atrophy as morphological changes in skeletal muscle. Applications of hypoxic exposure include intermittent hypoxic exposure and hypoxic training, both of which may lead to angiogenesis in a mechanism different from normal hypoxic exposure. In this review, we present some findings on the effects of hypoxia exposure on skeletal muscle and discuss whether satellite cells are involved in promoting angiogenesis by hypoxia.