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1 0 0 0 糖尿病における血管合併症

著者: 及川眞一
雑誌: 日本内分泌学会雑誌 (ISSN:00290661)
巻号頁・発行日: vol.76, pp.62-63, 2000-02-20

2020-10-07 11:40:14
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https://ci.nii.ac.jp/naid/10005553162

1 0 0 0 IR がんサバイバーの闘病体験と必要なケアリング

著者: 重久加代子
出版者: 国際医療福祉大学学会
雑誌: 国際医療福祉大学学会誌 = Journal of the International University of Health and Welfare (ISSN:21863652)
巻号頁・発行日: vol.25, no.2, pp.92-105, 2020-08-20

目的:がんサバイバーの闘病体験と必要なケアリングを明らかにすることである.方法:患者会で活動するがんサバイバー 5 名に闘病体験時の看護師の関わりについて半構造化面接を行い質的に分析した.結果:3 つの時期の 14 の状況より 71 の必要なケアリングと 14 の状況のケアリングが抽出された.〈がんの診断を受け入院するまでの時期〉では 2 の状況と【がんや検査に対する不安や苦痛を理解したケアリング】等である7 のケアリング,〈入院し治療を受ける時期〉では 7 の状況と【主体的な療養へのケアリング】,【全人的な理解と尊厳を守るケアリング】等である 36 のケアリング,〈外来での治療継続と経過観察の時期〉では 5 の状況と【在宅療養中の心身の苦痛とセルフケアへのケアリング】,【生き方や価値観を尊重したケアリング】等である 28のケアリングが抽出された.結論:これらは,本研究の定義である「対象者を大切な存在として認識し,その人の能力を最大限生かせるかかわり」を反映した,がん看護のケアリングになることが示された.

2020-10-07 11:24:56
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https://ci.nii.ac.jp/naid/120006882060

1 0 0 0 IR 準市場の優劣論と障害者福祉の選択制( 4・完)

著者: 児山正史
雑誌: 人文社会科学論叢 (ISSN:24323519)
巻号頁・発行日: no.9, pp.61-74, 2020-08-28

2020-10-07 11:24:29
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https://ci.nii.ac.jp/naid/120006885736

1 0 0 0 各種降圧薬の圧受容器反射におよぼす影響

著者: 相原彰子宗像正徳今井潤布川徹伊藤貞嘉吉永馨
雑誌: 日本災害医学会会誌 = The journal of Japan Accident Medical Association (ISSN:0386975X)
巻号頁・発行日: vol.46, no.11, pp.690-696, 1998-11
参考文献数: 16

2020-10-07 11:22:02
1 + 1 Wikipedia

https://ci.nii.ac.jp/naid/10007333537

1 0 0 0 本態性高血圧症の発症と進展

著者: 伊藤貞嘉
雑誌: ホルモンと臨牀 (ISSN:00457167)
巻号頁・発行日: vol.52, no.8, pp.823-827, 2004-08-01
参考文献数: 23

2020-10-07 11:22:02
1 + 1 Wikipedia

https://ci.nii.ac.jp/naid/10013547244

1 0 0 0 腎機能のパラクリン機構調節

著者: 伊藤貞嘉
雑誌: 日本内分泌学会雑誌 (ISSN:00290661)
巻号頁・発行日: vol.72, no.2, 1996-03-20

2020-10-07 11:22:02
1 + 1 Wikipedia

https://ci.nii.ac.jp/naid/10010971357

1 0 0 0 慢性腎不全の現状と対策

著者: 伊藤貞嘉
出版者: 一般社団法人日本内科学会
雑誌: 日本内科学会雑誌 (ISSN:00215384)
巻号頁・発行日: vol.91, 2002-02-20

2020-10-07 11:22:02
1 + 1 Wikipedia

1 0 0 0 炎症性疾患と低Na血症

著者: 太田昌宏伊藤貞嘉
雑誌: 臨床病理 (ISSN:00471860)
巻号頁・発行日: vol.46, pp.28, 1998-09-30
参考文献数: 5

2020-10-07 11:22:02
1 + 1 Wikipedia

https://ci.nii.ac.jp/naid/10005477358

1 0 0 0 レニン分泌の密集斑機構とプロスタグランジン

著者: 伊藤貞嘉阿部圭志尾股健保嶋実吉永馨
出版者: 社団法人日本腎臓学会
雑誌: 日本腎臓学会誌 (ISSN:03852385)
巻号頁・発行日: vol.30, no.1, pp.85-90, 1988

To examine the role of prostaglandins (PGs) in the macula densa mechanism of renin release, rabbit afferent arteriole (Af) alone and afferent arteriole with macula densa attached (Af+MD) were microdissected and incubated consecutively. Hourly renin release rate from a single Af (or Af+MD) was calculated and expressed as ngAI·h-1·Af-1 (or Af+MD-1)/h (where AI is angiotensin I). Basal renin release rate from Af was 0.84±0.14ngAI·h-1·Af-1/h (X±SEM, n=23) and remained stable throughout the incubations. Basal renin release rate from Af+MD was 0.33±0.04ngAI·h-1Af+MD-1/h (n=17), which was significantly lower (p<0.01) than that from Af. When furosemide (1.5 mM) was added to Af, no significant change in renin release rate was observed. However, when furosemide was added to Af+MD, renin release rate increased from 0.40±0.05 to 1.59±0.15ngAI·h-1·Af+MD-1/h (n=10, p<0.01). After the pretreatment with indomethacin, a cyclooxygenase inhibitor, furosemide still increased renin release rate from 0.17±0.02 to 0.56±0.09 ng AI·h-1·Af+MD-1/h (n=5, p<0.05) ; however, indomethacin pretreatment reduced both basal and furosemide-stimulated renin release rate (p<0.05). In the presence of PGI2 (10 μM), renin release rate from Af increased from 0.45±0.14 to 1.49±0.53 ng AI·h-1·AN/h (n=9, p<0.05), and further increased to 4.50±1.24 ng AI·h-1·Af-1/h (p<0.02) after removal from PGI2. When PGE2 (10 μM) was added to Af+MD, renin release rate increased from 0.54±0.09 to 1.26±0.24ng AI·h-1Af+MD-1/h (n=8, p< 0.05). However PGE2 had no effect on renin release rate from Af alone. We concluded that (1) the prostaglandin system may be a modulating factor of response in the macula densa mechanism of renin release, (2) PGI2 has direct action on renin release from affer-ent arteriole, and (3) PGE2 may participate in the control of renin release through the action on the macula densa.

2020-10-07 11:22:02
1 + 1 Wikipedia

1 0 0 0 白ウサギ腎表層,中皮質,傍髄質輸入細動脈のレニン含量

著者: 主代昇阿部圭志伊藤貞嘉三沢誠一吉永馨
出版者: 社団法人日本腎臓学会
雑誌: 日本腎臓学会誌 (ISSN:03852385)
巻号頁・発行日: vol.34, no.10, pp.1107-1111, 1992

Tissue renin content within the kidney decreases from outer to inner cortex. However, it is not known whether this gradient is due to a decrease in the number of afferent arterioles from the outer to inner cortex or the decrease in renin content per afferent arteriole. Furthermore, it is still controversial whether sodium depletion increases or decreases this gradient. According to Taugner et al., sodium depletion induces the extension of renin positive part of afferent arterioles from vascular pole toward interlobular artery. Since the length of extension may differ among superficial, midcortical, and juxtamedullary afferent arterioles, the observed gradient may vary depending on whether the entire afferent arteriole or only the vascular pole is examined. In the present study, we microdissected the entire afferent arterioles from superficial, middle, and juxtamedullary cortex of rabbit kidney, and examined tissue renin content. We studied: 1. whether tissue renin content per afferent arteriole decreases from the outer to inner cortex. 2. whether sodium depletion affects the gradient of tissue renin content within the cortex. In result, we reached the conclusions, as follows: 1. Tissue renin content per afferent arteriole decreases steeply from superficial to midcortical to juxtamedullary afferent arterioles. 2. The absolute difference in renin content among the three types of afferent arterioles becomes greater during sodium depletion. The internephron heterogeneity of tissue renin content may contribute to functional heterogeneity.

2020-10-07 11:22:02
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1 0 0 0 腎疾患を伴った高血圧 (特集高血圧治療のストラテジー) -- (合併症をもつ患者への降圧療法)

著者: 大高亮彦伊藤貞嘉
出版者: 南山堂
雑誌: 治療 (ISSN:00225207)
巻号頁・発行日: vol.82, no.4, pp.1339-1346, 2000-04

2020-10-07 11:22:02
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https://ci.nii.ac.jp/naid/40002457414

1 0 0 0 腎輸入細動脈におけるアデノシンの作用

著者: 矢尾板啓伊藤修有馬秀二遠藤好美竹内和久尾股健伊藤貞嘉
出版者: 社団法人日本腎臓学会
雑誌: 日本腎臓学会誌 (ISSN:03852385)
巻号頁・発行日: vol.41, no.7, pp.697-703, 1999

We investigated the direct effect of adenosine on afferent arterioles (Af-Arts) and the receptor subtype that mediates the constrictor or dilator action of adenosine . Af-Arts were isolated from the superficial cortex of rabbit kidney and perfused in vitro. Adenosine added to either the lumen or bath constricted the Af-Arts in a dose dependent manner. This constriction was blocked by the A1 receptor antagonist, 6-oxo-3- (2-phenylpyrazole (1, 5-a) pyridin-3-yl) -1(6H) -pyridazinebutyric acid (FK838) or 8-cyclopentyl-1, 3 - dipropylxanthine (DPCPX). We also examined the effect of adenosine on preconstricted Af-Arts with norepinephrine. Adenosine added to either the lumen or bath further constricted the preconstricted Af-Arts. In the presence of FK838, adenosine added to either the lumen or bath dilated the preconstricted Af-Arts, but in a different dose dependent manner. Adenosine induced dilation was inhibited by the A2 receptor antagonist, 3, 7-dimetyl-1- propargylxanthine (DMPX). These data indicate that adenosine constricts Af-Arts via A1 receptors and that adenosine dilates preconstricted Af-Arts via A2 receptors when A1 receptors are blocked.